[Microbiology] Atlas of Vibrio, Aeromonas, Plesiomonas, and Campylobacter Species

Vibrio, Aeromonas, Plesiomonas, and Campylobacter Species, Atlas of Vibrio, Aeromonas, Plesiomonas, and Campylobacter Species, MICROBIOLOGY ATLAS, SUBCLINICAL ATLAS, atlas in medical, tuyenlab.net

Fig 1. A, Microscopic morphology of Vibrio sp. on Gram-stained smear.
B, Acridine orange stain of Vibrio cholerae.

Fig 2. O/129 Susceptibility test for Vibrio sp. Left, Resistant; right, susceptible.

Fig 3. The action of cholera toxin. The complete toxin is shown binding to the
GMI-ganglioside receptor on the cell membrane via the binding (B) subunits. The active
portion (A1) of the A subunit catalyzes the adenosine diphosphate (ADP)–ribosylation of
the Gs (stimulatory) regulatory protein, “locking” it in the active state. Because the G
protein acts to return adenylate cyclase from its active to inactive form, the net effect is
persistent activation of adenylate cyclase. The increased adenylate cyclase activity results
in accumulation of cyclic adenosine 3’ 5’-monophosphate (cAMP) along the cell
membrane. The cAMP causes the active secretion of sodium (Na), chloride (Cl−),
potassium (K+), bicarbonate (HCO3 −), and water out of the cell into the intestinal lumen.

Fig 4. Vibrio cholerae on thiosulfate citrate bile salts
sucrose agar.

Fig 5. Gram stain of Aeromonas.

Fig 6.  Aeromonas hydrophila exhibiting β-hemolysis
on sheep blood agar.

Fig 7. Aeromonas caviae exhibiting lactose
fermentation on MAC agar.

Fig 8. Both Aeromonas and Plesiomonas spp. will
grow in nutrient broth with 0% NaCl (left), whereas neither
will grow in nutrient broth with 6% NaCl (right).

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